Post-Mortem Pathology (Paperback)

This historic book may have numerous typos and missing text. Purchasers can download a free scanned copy of the original book (without typos) from the publisher. Not indexed. Not illustrated. 1906 Excerpt: ...number of cells present. This atrophy may follow chronic emphysema, phthisis, chronic disease of the kidney, or starvation. Lymphoid marrow is gray red or dark red, according to the amount of blood it contains. In pernicious anaemia the marrow of the long bones resembles raspberry jelly, while in leukaemia it has a flesh-pink to a gray-yellow color, like that of pus. Fatty degeneration occurs in the cells and capillaries of the marrow, sometimes with necrotic foci, in cases of typhoid, typhus, and relapsing fever. In osteomyelitis there is often a purulent inflammation, frequently complicated by transformation of the marrow into a vivid red, tough, fibrinous material, and with effusion into the cavity of a joint. Necrotic changes in the bone follow. Hypertrophy of the marrowcells is seen in oligaemia, leukaemia, chronic pulmonary tuberculosis, chronic suppurative osteitis, cancerous cachexia, typhoid fever, croupous pneumonia, septic affections, acute endocarditis, and smallpox; while hypertrophy of the fatty tissue occurs in cases of general atrophy of the skeleton, sometimes involving the entire bone. Necrosis.--Necrosis arises as the result of shutting off of the blood-supply. It follows infective embolus, injury, poisoning, as from phosphorus, and as a sequela of scrofula and the infective fevers. Caries, necrosis superficialis, or erosion is, as a rule, circumscribed, but may be diffuse or phagedenic. It occurs in the cancellous extremities of a bone, usually in the tibia, femur, humerus, phalanges, skull, lower jaw, clavicle, and ulna, and affects the joints secondarily. It is always associated with periostitis, osteitis, or osteomyelitis. It is generally dry and anaemic, but in cases of sudden onset may be moist. The bone is ulcerated or worm-eaten i...