This historic book may have numerous typos and missing text. Purchasers can download a free scanned copy of the original book (without typos) from the publisher. Not indexed. Not illustrated. 1841 Excerpt: ...tint results fiom more complete combination of the principles of the blood with the renal tissue, and not from a bloodless condition of the part; an opinion against which the objection already made, and obviously applicable during the first stage, now ceases to bear. In the third, the hyperaemia disappears more completely, the mottled aspect is lost, and a uniform slightly yellowish tint prevails; anaemia is now, according to M. Rayer, general. But, as M. Solon well observes, the term anaemia, which may be very correctly employed in speaking of the state of the kidneys in subjects dying of hemorrhage, is not applicable, in point of accurate description, to the discoloration now referred to. And M. Rayer himself elsewhere lends force to this observation; for he points out (p. 324) its yellow tint as actually distinguishing this condition of the renal tissue from the anaemic kidneys of certain tuberculous subjects. In the fourth phasis the previous state of discoloration still prevails, but the deposition of "Bright's granulations" marks it distinctly; the fifth seems to be a mere modification of this stage. In the sixth, the granulations commonly disappear, and differing in this from all its predecessors, this stage is frequently marked by a tendency to congestion, contraction, or atrophy on the part of the kidney. M. Solon, it will be seen, carries us a step further, and attempts to range all renal, analogous, and heterologous products as an ordinary and necessary sequence of Bright's disease, --a proceeding so utterly irreconcilable with what is known respecting those products, and with the general laws of pathology, that we can only marvel at its adoption. The anatomical characters of the affection claims our first attention, and in order to exhi...